Oncometabolite 2-hydroxyglutarate is a competitive inhibitor of α-ketoglutarate-dependent dioxygenases.

نویسندگان

  • Wei Xu
  • Hui Yang
  • Ying Liu
  • Ying Yang
  • Ping Wang
  • Se-Hee Kim
  • Shinsuke Ito
  • Chen Yang
  • Pu Wang
  • Meng-Tao Xiao
  • Li-xia Liu
  • Wen-qing Jiang
  • Jing Liu
  • Jin-ye Zhang
  • Bin Wang
  • Stephen Frye
  • Yi Zhang
  • Yan-hui Xu
  • Qun-ying Lei
  • Kun-Liang Guan
  • Shi-min Zhao
  • Yue Xiong
چکیده

IDH1 and IDH2 mutations occur frequently in gliomas and acute myeloid leukemia, leading to simultaneous loss and gain of activities in the production of α-ketoglutarate (α-KG) and 2-hydroxyglutarate (2-HG), respectively. Here we demonstrate that 2-HG is a competitive inhibitor of multiple α-KG-dependent dioxygenases, including histone demethylases and the TET family of 5-methlycytosine (5mC) hydroxylases. 2-HG occupies the same space as α-KG does in the active site of histone demethylases. Ectopic expression of tumor-derived IDH1 and IDH2 mutants inhibits histone demethylation and 5mC hydroxylation. In glioma, IDH1 mutations are associated with increased histone methylation and decreased 5-hydroxylmethylcytosine (5hmC). Hence, tumor-derived IDH1 and IDH2 mutations reduce α-KG and accumulate an α-KG antagonist, 2-HG, leading to genome-wide histone and DNA methylation alterations.

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عنوان ژورنال:
  • Cancer cell

دوره 19 1  شماره 

صفحات  -

تاریخ انتشار 2011